Nicotinamide Mononucleotide (NMN) is an activated form of B3 and is a highly bioavailable nucleotide. NMN is increased by NAD+ which increases the body’s main energy currency (ATP), balances the circadian rhythm, and enables hundreds of enzymatic reactions – many of which delay ageing. NAD+ levels naturally decline with age, slowing down the speed and accuracy of DNA repair, and impairing energy utilization capabilities at the cellular level.
By boosting NAD+ levels, NMN helps combat these age-related symptoms, promoting healthy ageing and longevity. NMN will help you to operate at your fullest potential, keeping you on track towards a longer and stronger life.
NMN may improve.
Increase cell activity
Boosts NAD+ Levels
Promote healthy ageing
Increase energy levels
Supports cognitive function
Relieve fatigue
Enhance brain function
Assist in anti-ageing and longevity
Critical in DNA repair and cell survival
Enhances Mitochondrial Health After Oxidative Stress
research & studies on NMN.
Various studies has been completed by multiple Universities, Research companies and scientists globally. We are made it easy for you to have some of these studies all in one place.
David Sinclair, PhD, AO, is a professor of genetics at Harvard Medical School. One of the leading innovators of his generation, he has been named by Time as “one of the 100 most influential people in the world” and top fifty most influential people in healthcare. He is a board member of the American Federation for Aging Research and has received more than thirty-five awards for his research and major scientific breakthroughs.
Nicotinamide Mononucleotide: Exploration of Diverse Therapeutic Applications of a Potential Molecule – More
Effect of oral administration of nicotinamide mononucleotide on clinical parameters and nicotinamide metabolite levels in healthy Japanese men –More
Nicotinamide mononucleotide preserves mitochondrial function and increases survival in hemorrhagic shock –More
Nicotinamide mononucleotide, an intermediate of NAD+ synthesis, protects the heart from ischemia and reperfusion – More
